Deleting Ku70 is milder than deleting Ku80 in p53-mutant mice and cells

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Deletion of Individual Ku Subunits in Mice Causes an NHEJ-Independent Phenotype Potentially by Altering Apurinic/Apyrimidinic Site Repair

Ku70 and Ku80 form a heterodimer called Ku that forms a holoenzyme with DNA dependent-protein kinase catalytic subunit (DNA-PKCS) to repair DNA double strand breaks (DSBs) through the nonhomologous end joining (NHEJ) pathway. As expected mutating these genes in mice caused a similar DSB repair-defective phenotype. However, ku70(-/-) cells and ku80(-/-) cells also appeared to have a defect in ba...

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ژورنال

عنوان ژورنال: Oncogene

سال: 2009

ISSN: 0950-9232,1476-5594

DOI: 10.1038/onc.2009.57